Splenic clearance of rigid erythrocytes as an inherited mechanism for splenomegaly and natural resistance to malaria
Abstract
The sensing of poorly deformable red blood cells by the human spleen can be mimicked in vitro.
Abstract
Retention of Plasmodium falciparum ring-infected erythrocytes in the slow, open microcirculation of the human spleen.
Abstract
Ex vivo perfusion of human spleens maintains clearing and processing functions.
Abstract
High-throughput microsphiltration to assess red blood cell deformability and screen for malaria transmission-blocking drugs.
Abstract
Plasmodium falciparum STEVOR proteins impact erythrocyte mechanical properties.
Abstract
A switch in infected erythrocyte deformability at the maturation and blood circulation of Plasmodium falciparum transmission stages.
Abstract
Microsphiltration: a microsphere matrix to explore erythrocyte deformability.
Abstract
Anti-Self Phosphatidylserine Antibodies Recognize Uninfected Erythrocytes Promoting Malarial Anemia.
Abstract
Malaria induces anemia through CD8+ T cell-dependent parasite clearance and erythrocyte removal in the spleen.
Abstract